This one food can protect men from cancer

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This one food can protect men from cancer

Studies about micronutrients and cancer generally yield positive results. 

Getting the proper amounts of minerals, vitamins, and fatty acids can undeniably influence cancer.

And some of these food components – such as linoleic acid, vitamin E, and beta-carotene – can have quite powerful effects, making them stand out from the rest.

Selenium, and specifically selenomethionine, is one such micronutrient. 

Selenomethionine is an all‐natural amino acid found in plants and animals.

It is essentially identical to methionine – an amino acid with antioxidant activity that is involved in metabolism and other aspects of health. 

The difference with selenomethionine is that the sulfur atom is replaced by selenium, giving it radically different properties. 

And this is actually the form in which selenium is generally found in food. So we do consume it in small amounts every day. 

We need selenium in order to form some enzymes involved in thyroid hormone regulation. It also provides antioxidant activity. 

So it’s beneficial in two ways.

But, as a relatively rare amino acid, it’s sometimes helpful to supplement selenium. 

Unfortunately, the foods containing the highest levels – such as Brazil nuts and soy – are also high in polyunsaturated fatty acids (PUFAs), and immunogenic proteins. 

And soy is also high in phytoestrogens and polyamines.

But selenomethionine could be worth supplementing in light of its anticancer effects. 

Not many would consider it ideal to have too many supplements crowding the cabinets, and most nutrients should come from food.

But selenomethionine is certainly one of the few worth buying.

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The most well‐known selenium study is this one. 

Up to this point, it was known that selenium – just the lone mineral – was slightly protective against cancer. 

This is because the results of meta‐reviews had shown pretty consistent findings. 

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But in 1996, some researchers decided to investigate selenium further… 

They recruited quite a few people for the study – more than 1,000 – and split that bunch of people into two groups of 650. 

One group was supplemented with 200 μg (micrograms) of selenium and the other was not.

The results were fairly striking, to say the least. 

The risk ratios for all cancers measured were far below unity (see pink column):

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Lung cancer was reduced by one‐third, prostate cancer by two‐thirds, and colon cancer had dropped down to 14% that of the controls.

To put it another way… For every eight people who got colon cancer, seven of them were NOT taking selenium. 


“No cases of selenium toxicity occurred.” (Clark, 1996)

So what to make of this? Do these anti‐cancer effects have anything to do with thyroid hormone? 

Probably not, but it gets even more interesting than that. 

Even though thyroid hormone does rise a bit with 200 μg selenomethionine, it’s not as dramatic a result as the previous study had with cancer. 

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While active thyroid hormone (T₃) does increase the metabolic rate, among other benefits, you wouldn’t expect much of a boost from this slight rise.

In this study as in the earlier one, the participants received the same dose (200 μg).

And the changes in plasma selenium were similar (76 ng/mL in the earlier study versus 90 ng/mL in this one) – proving similar absorption.

Back then, investigators had no idea of how this worked. 

They didn’t seem to make the distinction between selenium and selenomethionine – and spoke of them interchangeably.

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But the anticancer effect is actually now understood. 

And this is something that plain selenium atoms cannot do by themselves. 

You need selenomethionine, specifically, to inhibit cancer to this degree.

Selenomethionine interferes with polyamine synthesis, and safely.

Polyamines are small molecules involved in cell proliferation. 

And they are so involved as to be nearly synonymous with it:

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A few years after the classic study, a few sharp biochemists put two and two together and decided to test selenomethionine on polyamine synthesis. 

Polyamines are small molecules that control the rate of DNA synthesis directly.

The logic is clear…

Methionine is necessary to create the polyamines involved in cell proliferation and cancer

And selenomethionine, a very similar molecule, powerfully inhibits cancer.

So this leads to the idea that selenomethionine could be inhibiting polyamine synthesis by competing with methionine

Polyamines rise and fall in ways that mirror the cell cycle.

“As polyamines are clearly implicated in cell cycle progression, it was not surprising to find the decrease in polyamines from selenomethionine administration…” (Redman, 1997)

The researchers noticed a decrease in growth and polyamine synthesis with selenomethionine.

No other amino acid can do this… Not even L‐threonine is this powerful.

Threonine can become methylglyoxal and, theoretically, will help…

But it doesn’t appear to be as effective as selenomethionine gram‐for‐gram.

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And to further prove how it works, they showed a reversal with added polyamines.

The cancer cells could be made to proliferate again, quite predictably, with additional polyamines. 

“In the present study we also noted that selenomethionine administration resulted in mitotic arrest in both tumor cell lines.” (Redman, 1997)

The involvement of polyamines in cancer had been noted as early as the 1920s by William Koch.

And as early as the 1960s, they were found to bind very strongly to DNA. 

Polyamines can actually change DNA from the right‐handed A‐configuration to the left‐handed Z‐configuration, powerfully increasing the replication rate (Thomas, 1991). 

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This effect has even been noted during PCR amplification, the routine replication of DNA outside of the body for criminal, genetic, or biochemical investigation.

The main enzyme that creates polyamines, ornithine decarboxylase, is upregulated by both androgens and prostaglandin E₂…

These are the two hormones most associated with prostate cancer growth. 

So it seems as if every path leads to polyamines. 

And selenomethionine is safe. 

The body doesn’t distinguish between methionine and selenomethionine, so they both get incorporated into proteins at roughly the same rate. 

Plants also cannot make this distinction. 

So it basically seems to be almost just like methionine, but better. 

I think it’s fair to think of it as “preferred methionine.”

Interestingly, the Japanese do have seven times the amount in their muscle tissue as Americans. 

(Could that be because they eat so much fish in Japan?)

“The selenium contents of human skeletal muscle reflect accordingly the dietary selenomethionine intakes and were found to be the highest in Japanese adults (1,700 ng/g), followed by Canadians (370 ng/g) and Americans (240 ng/g), with the lowest (61 ng/g) found in New Zealand adults.” (Shrauzer, 2000)

Compare these high levels to the mere doubling of plasma levels seen at the small 200 μg dose. 

I think it’s fair to assume that taking a bit more than 200 μg would also be safe. The upper limit is set at 500 μg by the Institute of Medicine.

Perhaps selenomethionine could even be more effective yet, with an even higher dose?

And higher doses of 800 μg selenomethionine have been found to be safe, roughly tripling the plasma selenium levels. 

“‘Some of the subjects in this study ingested >800 μg selenium/d for 16 weeks. This is considerably more than the Institute of Medicine’s tolerable upper level of 400 μg/d. No signs of selenium toxicity (hair loss and nail changes) were observed, in agreement with Chinese observations in subjects with intakes of about 800 μg.” (Burk, 2006)

So, besides raising active thyroid hormone and increasing metabolism, selenomethionine can powerfully inhibit cancer. 

Coupled with L‐threonine, and a diet low in linoleic acid, you might think cancer could be nearly completely avoidable.

But in any case, selenomethionine has been shown to reduce the risk of colon cancer sevenfold.

And the study by Redman shows how important polyamines are for cell proliferation. 

For this reason, the polyamine precursors arginine, ornithine, and common S‐methionine, should probably be used with caution. 

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Matt Cook is editor-in-chief of Daily Medical Discoveries. Matt has been a full time health researcher for 26 years. ABC News interviewed Matt on sexual health issues not long ago. Matt is widely quoted on over 1,000,000 websites. He has over 300,000 daily newsletter readers. Daily Medical Discoveries finds hidden, buried or ignored medical studies through the lens of 100 years of proven science. Matt heads up the editorial team of scientists and health researchers. Each discovery is based upon primary studies from peer reviewed science sources following the Daily Medical Discoveries 7 Step Process to ensure accuracy.


Schrauzer, Gerhard N. "Selenomethionine: a review of its nutritional significance, metabolism and toxicity." The Journal of nutrition (2000)


Redman, Claire. "Involvement of polyamines in selenomethionine induced apoptosis and mitotic alterations in human tumor cells." Carcinogenesis (1997)


Combs, Gerald F. "Effects of selenomethionine supplementation on selenium status and thyroid hormone concentrations in healthy adults." The American journal of clinical nutrition (2009)


Clark, Larry C. "Effects of selenium supplementation for cancer prevention in patients with carcinoma of the skin: a randomized controlled trial." Jama (1996) 


Comstock, George W. "Serum retinol, beta-carotene, vitamin E, and selenium as related to subsequent cancer of specific sites." American journal of epidemiology (1992)