THEY don’t want you to know about this cheap, natural cancer cure

Vitamin C (Ascorbic Acid) from Oranges

Big Pharma doesn’t want us to know about this because it works better than their expensive treatments… And you can find it at the store for less than $1…

—-Important Message About Vitamin C—-

Here’s why Super Vitamin C is better than any other form of vitamin C…

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  • Stops cancer in its tracks…potentially life-saving…
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Here’s how to get this new SUPER Vitamin C today (and it’s free).

———-

THEY Don’t Want You to Know About This Cheap, Natural Cancer Cure

Vitamin C is perhaps the best-known vitamin, and could be neglected for this very reason.

Because it is so commonplace, people who imagine “the most expensive pharmaceuticals are necessarily the most effective” think of vitamin C as inferior.

Yet this is flawed thinking… Because synthetic drugs are developed and marketed primarily for profit and not necessarily for efficacy.

Some of these same people imagine that if a cheap cancer treatment was widely available everyone would know about it…

And then it would immediately be co-opted by the medical establishment for use against cancer.

But this is naïve thinking… It would be financial suicide for drug companies, hospitals, and most medical doctors.

Some of the most effective cancer treatments are cheap, safe, effective, and freely available…

And they are hiding in plain sight.

Baicalein, lapachol, gamma-tocopherol, threonine, selenomethionine, and ascorbyl palmitate are all very effective supplements…

But hardly anybody is trumpeting that horn.

These vitamins, amino acids, and phytochemicals are so cheap that not even chiropractors can make money off them…

The only people who benefit from their promotion are online retailers, supplement companies, and (of course) the patients themselves.

And if a patient chooses to have radiation, surgery, or immunotherapy, they can gain additional benefits through taking the right supplements while avoiding the wrong foods.

Plain old vitamin C has a role to play in the chemotherapy and chemo-prevention of cancer.

And we need it for collagen synthesis and immune function besides.

While this isn’t a magic bullet in itself, especially in advanced cases, it has been scientifically proven to inhibit metastasis.

And it has been documented to yield complete remissions.

This latter effect was published by Ewan Cameron and Linus Pauling in the 1970s:

In this article, Cameron reports a complete regression of lung cancer observed in a patient shortly after taking vitamin C (ascorbic acid).

Yet this was in no way a spontaneous remission…

The cancer came back upon cessation of the vitamin…

And then it disappeared again upon its re-institution.

Ewan Cameron seems to be trustworthy – and to have no motive besides scientific progress and patient well-being.

Cameron was a senior doctor and administrator of the Vale of Leven Hospital in Loch Lomondside, Scotland.

This was not an alternative hospital. It was a conventional one.

So you could rightly think he’d have more financial motive not to report these findings.

“Reduction in dosage some months later coincided with reactivation of the disease process. The re-institution of regular high-dose ascorbic acid therapy induced a second complete remission.”

Unlike most terminal cancer cases who are given unconventional treatments, this patient was not considered “untreatable” at the time of initiation.

This patient had not received any of the conventional treatments (surgery, radiation, cytotoxic drugs) – because of an administration delay having nothing to do with the prognosis.

The efficacy of most unconventional treatments, such as the Gerson therapy, is handicapped by its patients often being at very advanced stages.

That includes patients who have undergone radiation and/or chemotherapy before that point…

And then they’ve essentially been marked off as a “lost cause” by the conventional physicians and sent home.

This study implies that vitamin C would be even more effective on people who have not been previously irradiated or who show up fully metastasized.

The study documented all of this with X-rays and biopsies. So the presence of cancer cannot be passed off as a misdiagnosis.

Conventional doctors seem to enjoy overly scrutinizing reports of non-pharmaceutical remissions…

And this is in line with the notion that cancer is always fatal unless you do something heroic and spend a lot of money.

After all, it’s bad for business to let patients think they’d be better served at home.

“Re-institution of regular high-dose ascorbic acid supplementation induced a second more gradual but equally complete remission.”

By all accounts, this appears to have been a vitamin C-induced remission.

That was Cameron’s impression – and his conclusion from the initial remission was confirmed by the second remission.

The erythrocyte sedimentation rate, a biomarker for cancer, was perfectly correlated against the vitamin C intake:

As vitamin C intake went up, erythrocyte sedimentation rate decreased.

As of February 11, 1975, the patient remained “fit and well,” and also in “active heavy employment.”

By the time the study was published, the patient was taking 12.5 gm of vitamin C daily. And taking it diligently this time to avoid any chance of relapse.

Twelve grams seems like a lot, but it is perfectly safe…

Indeed, people have taken 20 gm per day without any side effects worth concern.

The best estimates of the ideal intake is around 3 gm per day for healthy people…

That figure was extrapolated from the production rates of animals that can make their own vitamin C (Pauling, 1970).

Also, a similar daily intake recommendation can be gathered through estimating the vitamin C content of primitive human diets.

While the RDA of 60 mg per day can barely prevent scurvy, this intake results in plasma vitamin C levels far below that of all other animals – and humans eating naturally.

Yet, because humans are more intelligent, we ought to do better than the other animals…as long as we don’t let our routine eating habits get too unnatural.

There’s good reason to believe this one remission was not a fluke, as many others have been reported from vitamin C.

The same doctors actually treated hundreds of cancer cases this way.

The results of the first 50 cases are in the report below:

The only difference between these patients and the previous case report is that these patients were all considered “untreatable” from the outset.

Naturally, you cannot expect a 100% remission rate… So far I have seen nothing that works in every advanced case of cancer.

Yet it did do fairly well, as evidenced by the 10% full remission rate.

This is considerably greater that the rates of spontaneous remission..

And it would certainly be much higher if the cancer in these patients hadn’t already been so advanced.

And it noticeably helped the majority of patients (58%) despite their dire condition.

However, for 8% it “helped” too much…

These patients’ tumors actually lysed too quickly after taking 10 gm of vitamin C per day – they died of hemorrhage and/or shock from autolysis.

Too much tissue breakdown too quickly can lead to hyperammonemia, often fatal in itself.

So it’s probably a good idea to take it slow at first, just in case…

…perhaps by starting with the nominal figure of 3 gm per day and then working up.

William Koch also noted that in his patients tumors can be lysed too quickly for comfort.

This was back in the ’30s while using methylglyoxal along with dietary modifications.

Yet there is every indication that vitamin C helps the majority of people (with cancer or not).

And there are many plausible biochemical reasons why you would expect it to.

First of all, vitamin C significantly inhibits metastasis.

This was experimentally demonstrated in rats and explained by the tumors becoming encapsulated by collagen, thereby restraining their growth (Cha, 2012).

“Tumors from ascorbate-supplemented mice showed diffuse staining of collagen IV around the core and a robust collagen I tumor capsule. Scorbutic mice tumors demonstrated a strong staining of collagen IV around the core, within the tumor, and weaker, disorganized, or missing collagen I tumor capsule.”

Vitamin C is a prerequisite for collagen synthesis. We need it to physically stabilize the fibrils by hydroxylating prolyl and lysyl residues.

Natural amounts of vitamin C will lead to proper synthesis of collagen…

Collagen is a structural protein the body uses for many things.

And not the least of those is to encapsulate malignant tumors to slow their growth.

Yet vitamin C also stimulates natural killer cells, and quite dramatically at that (Heuser, 1997).

Natural killer cells were discovered in the late ’70s.

Scientists noticed their ability to recognize abnormal cells (such as mutated cells and virus-infected cells) and then destroy them with maximum prejudice.

Of the 10 or so individual types of immune cell, natural killer cells remain the only type known to significantly inhibit cancer.

Scientists didn’t know about these cells in the early- to mid-1970s.

And maybe that is why Cameron and Pauling explained vitamin C’s beneficial effect through collagen encapsulation and hyaluronidase inhibition.

Yet hyaluronidase is certainly a powerful cancer enzyme. It is capable of depolymerizing hyaluronic acid of the extracellular matrix.

This enzyme is correlated with the invasive potential of Candida species and also the metastatic potential of cancer.

Although vitamin C is a weak hyaluronidase inhibitor at about 6 mM, it’s palmitate ester can do it at a far lower concentration – 4.2 μM.

Vitamin C palmitate is a veritable hyaluronidase inhibitor and also widely available.

This supplement has the additional benefit of being distributed more into fatty tissues such as the nervous system, sex organs, and adipose tissue.

And we have our own “hyaluronidase inhibitor” that our bodies produce. It’s a glycoprotein abbreviated as “PHI.”

There are good reasons to think vitamin C is necessary for the production of endogenous PHI in some way…

Most likely it does this by becoming a structural unit of its polysaccharide chain (Pauling, 1973).

Although inhibiting metastasis is very important, metastasis inhibition is not the same thing as regression.

That would be better termed “growth retardation” and/or “cytostasis.”

Those are the two terms Cameron used to describe “grade 3 & 4 beneficial responses” to vitamin C.

Vitamin C’s ability to cause growth retardation (the “standstill effect”) was more common than regression in his studies.

While only about 10% of patients fully regressed with vitamin C use, another 44% were notably improved by it.

The complete regressions are better explained in other ways, such as through the inhibition of HIF-1-alpha:

Hypoxia-inducible factor 1-alpha (HIF1A) is a transcription factor upregulated under oxygen deprivation.

It transcribes for the growth-promoting genes: VEGF, GLUT-1, BNIP3, etc.

Of these, vascular endothelial growth factor (VEGF) is probably the most notorious.

This growth factor greatly increases neogenesis.

And that is why tumors have so many blood vessels leading into them.

An increased blood supply is the natural response to hypoxia, or a lack of oxygen…

Yet when HIF-1-alpha is expressed in tumors, it ultimately allows them to create more blood vessels than in the surrounding areas.

Vascular endothelial growth factor (VEGF) is how cancer taps into the host’s supply of oxygen, protein, and glucose.

Yet access to this is also ensured by HIF1A’s concomitant transcription of GLUT-1, a glucose transporter protein.

This makes HIF1A a prime target in the fight against cancer.

However, this mediator is under control of a few other things besides oxygen.

Methylglyoxal has been shown to be capable of causing the degradation of HIF1A.

And we can understand this by realizing that it’s a biochemical signal for sufficient glucose.

Methylglyoxal is produced as a function of glucose flux, easier to sense than glucose on account of its reactivity.

HIF1A can be activated by hydrogen peroxide. That is often a signal of iron excess and/or low antioxidants.

This is why selenium can also prevent HIF1A activation, as we need selenocysteine for glutathione peroxidase.

Glutathione peroxidase is the “other hydrogen-peroxide-degrading enzyme” of the cells – besides catalase.

“Activation of the transcription factor HIF-1 allows solid tumors to thrive under conditions of metabolic stress. Because HIF-1 is switched off by hydroxylation reactions that require ascorbate, inadequate intracellular ascorbate levels could contribute to HIF-1 over-activation.”

Vitamin C works as an antioxidant in the lab (in vivo) in physiological concentrations.

So it could work by preventing HIF1A activation.

Yet its effects have been proven to extend beyond that…

Vitamin C is the cofactor needed for all prolyl-hydroxylase enzymes, including a specific isoform that hydroxylates HIF1A at proline 564, marking it for degradation (Jaakkola, 2001).

So vitamin C acts physically to degrade HIF1A, perhaps understood by noting its importance within the body.

Although our cells fundamentally need oxygen and glucose more than vitamin C, without it they can’t even produce working collagen.

Thus, it somewhat makes sense that HIF1A should also be regulated by vitamin C.

Without this vitamin, the cell will produce more VEGF to vascularize. (As discussed above, that’s about creating blood vessels.)

And this vitamin definitely decreased the concentrations of HIF1A.

This study was the first to show that HIF1A is inversely correlated with vitamin C in vivo, in humans.

And it was the first to show that HIF1A is also upregulated in the more invasive tumors.

The researchers measured both HIF1A and VEGF (among other things) in tumors taken from patients, as well as GLUT-1.

And this all what you’d expect based on previous studies…

As shown by Jaakkola earlier, vitamin C downregulated HIF1A.

And the classic proteins transcribed through HIF1A were all predictably downregulated as well.

Without VEGF, tumors are much less capable of thickening existing blood vessels and creating newer ones.

And without GLUT-1, a glucose transporter, the tumors would have a lower energy flux – which would attenuate their high proliferation rates.

And also, a lowered glucose flux means less cancer growth, greater host survival, and more regressions.

They even confirmed the longstanding observation that tumors (especially the advanced ones) have lower vitamin C levels than surrounding tissue.

This is in line with previous observations of low plasma ascorbate in cancer cases (Bodansky, 1952).

To summarize, here are the mechanisms through which vitamin C helps:

  • It encapsulates tumors with collagen
  • And that stimulates natural killer cells
  • It inhibits hyaluronidase
  • And it helps in yet another way by degrading HIF1A

While no “magic bullet” by itself, evidence shows that this vitamin is powerful enough to induce complete regressions in 10% of very advanced cases of cancer.

Plus it reliably inhibits metastasis, a good enough reason by itself to supplement or eat more fruit.

“Low ascorbate levels were also associated with elevated VEGF, GLUT-1, and BNIP3 protein levels and with increased tumor size, and there was a significant association between low tissue ascorbate levels and increased activation of the HIF-1 pathway.”

—-Important Message—-

This natural white powder can cure cancer and costs less than $1

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Big Pharma doesn’t want you to know about this natural powder because it works better than their expensive treatments.

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———-


Matt Cook is editor-in-chief of Daily Medical Discoveries. Matt has been a full time health researcher for 26 years. ABC News interviewed Matt on sexual health issues not long ago. Matt is widely quoted on over 1,000,000 websites. He has over 300,000 daily newsletter readers. Daily Medical Discoveries finds hidden, buried or ignored medical studies through the lens of 100 years of proven science. Matt heads up the editorial team of scientists and health researchers. Each discovery is based upon primary studies from peer reviewed science sources following the Daily Medical Discoveries 7 Step Process to ensure accuracy.
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