Two for one? Can this simple prostate tweak increase erections and cause you to grow a full head of hair?
Why Men Lose Their Hard-Ons
You know how it goes… Men who were roaring to go, multiple times a night, but then their penis stopped working.
It is flat, it is flaccid… that thing is dead…
Why do men go flat?
Men go flat because of high estrogen…
Their T level is okay, but their estrogen is sky-high. That’s what happened to me…and you know where that excess estrogen comes from?
Fat cells. That’s what creates all that extra erection-killing estrogen.
Luckily, I found this simple formula to youthify my body, melt away belly fat, and MOST importantly, get back fantastic erections. It lowers estrogen.
Yes, it’s a little bitter-tasting, but there is ZERO “cutting calories, counting carbs, or missing meals”… And my wife loves the new me, LOL.
Huh? Your prostate grows hair on your head?
Many studies have demonstrated a link between prostate cancer and hair loss.
This correlation is stronger than the one between prostate cancer and cardiovascular disease…
In fact, this link has been demonstrated in all studies that have looked for it…
Except for one study that assessed hair loss based on frontal yearbook photos alone (no attempt was made to quantify vertex – top of the head – hair loss).
On the other hand, all the truly thorough studies have shown an obvious correlation.
Finding the common denominator between hair loss and prostate cancer should help us understand the cause of both.
Following up on smaller studies which had shown a correlation, a group of Australians conducted a much larger study.
This was a study group of considerable size…
There were 1,497 cases and 1,434 control subjects.
The researchers found a significant correlation which was also dependent on severity – the people with the worst cases of prostate cancer also had the most hair loss.
“For high-grade disease, the odds ratios for all types of androgenetic alopecia were elevated, nearly 3-fold for vertex balding…”
These are strong correlations that are made even more significant by the large size of the study.
This effect has been found everywhere it has been looked for…
America, Australia, Korea, the Netherlands, and France.
This study found the same correlation. It was conducted in Europe with 669 participants.
And these subjects were young, all of them under 20.
This all but rules out most of the possible confounders. (A confounder is an unobserved exposure that could affect outcomes.)
The fact that this correlation is observable across ages, and across geographic locations, suggests that it represent a very real effect.
“When compared with control group subjects, those with prostate cancer were twice as likely to have signs of alopecia at age 20.”
So how do they explain it?
Well, they just mumble something about androgens, of course…
Even though testosterone has actually been shown to stimulate hair growth in vitro…
And finasteride doesn’t slow prostate cancer – at all.
In fact, the only significant finding from the use of finasteride in the study below was actually an increase in prostate cancer:
Caught up in the prostate cancer–androgen paradigm, these researchers decided to conduct a study into just that…
Men with prostate cancer and some without prostate cancer were given finasteride for one year.
The results were measured by biopsy and then compared with a biopsy taken a year before.
The slides were coded (meaning it was a blind study, removing bias from the results).
“Moreover, in patients with pre-existing prostatic intraepithelial neoplasia (PIN) lesions, treatment with finasteride was statistically significantly associated with an increased incidence of prostate cancer at 1 year.”
So an increase in prostate cancer from finasteride.
If androgens were responsible for prostate cancer, then finasteride should be the wonder drug.
But it’s not. Finasteride had the opposite effect in this study.
It’s true that lowering androgens can lead to a decrease in prostate volume – since androgens are always anabolic and the prostate is an androgen‐dependent organ.
But this fails to explain cancer – since volume does not equal cancer.
Cancer is cancer. And smaller people – with less volume – still get cancer.
If androgens were truly the cause…
There would be no need for terms such as ‘‘androgen‐refractory prostate cancer.”
That phrase tacitly admits that androgens are not a primary factor, while also implying that people just assume they ought to be.
Pinning every sex difference on androgens is just lazy. And women don’t even have prostates.
“This study raises serious questions about the probable efficacy of finasteride in preventing prostate cancer.”
Androgens cannot explain the geographic incidence either.
Men everywhere have essentially the same amount of androgens, yet vary wildly in prostate cancer rates.
Generally, you see a trend in prostate cancer the further north you go.
Genetics can’t explain this. Androgens can’t explain this.
“These geographic patterns are not readily explicable by other known risk factors for prostate cancer.”
More realistic risk factors probably involve vitamin D…
Or perhaps a dietary factor such as linoleic acid.
The further plants are from the equator, the more linoleic acid they synthesize.
This is probably because unsaturated fatty acids have a low freezing point, helping the plants resist colder climates.
Northerners eat more grains because grains are suitable to be grown northern climates.
While it’s true that we could all probably use a bit more vitamin D…
Prostaglandins derive solely from one dietary source – linoleic acid.
Prostaglandin D₂ is nearly synonymous with hair loss, and linoleic acid has been shown to accelerate prostate cancer in vitro.
And linoleic acid has been found to correlate directly with prostate cancer, to a very high degree.
In this classic study, Paul Godley compared prostate cancer to individual fatty acids.
This wasn’t just a flaky dietary recall study. This team actually took biopsies of adipose tissue.
“Animal studies have suggested that omega-6 fatty acids found in vegetable oils may promote prostate cancer.”
They determined which fatty acids were present within the body and in what ratios.
Then they correlated that information with prostate cancer…
They found unbelievably high-risk ratios.
The observed correlations were so strong that you’ll forget all about androgens.
This was observed exclusively with one fatty acid: linoleic acid…
Which happens to be the only fatty acid that acts as a precursor to prostaglandins.
“In Table 4, only linoleic acid demonstrated a statistically significant increase in risk.”
Consider also the in vitro demonstrations by Rose, who observed cancer cell growth with linoleic acid…
And you’ve essentially found the #1 risk factor for prostate cancer.
Luis Garza, among others, has shown that prostaglandin D₂ is highly correlated with hair loss in every way.
The common denominator appears to be linoleic acid.
And linoleic acid is what prostaglandins are made from.
“…a possible relationship between the rising breast cancer risk and an increase in the consumption of vegetable oils rich in linoleic acid (found in vegetable oil). This may also apply to prostate cancer.”
Considering the research on prostaglandins alone, we could actually expect hair loss to be correlated with prostate cancer, just as observed.
Prostaglandins can be considered hormones.
They are lipid signallers that tell a cell it’s under attack.
These signal the receptors in the nucleus, telling them to create more fat-metabolizing enzymes to help get rid of excessive prostaglandins.
But this shifts metabolism away from natural metabolism, and towards a pathological state of affairs.
Obviously, before we knew how to make fire, humans mostly lived in the tropics – where linoleic acid is scarce.
And the few exceptions who lived in the North, such as the Neanderthals…
…probably had enough sense not to use canola oil.
“Linoleic acid consumption (in vegetable oil) was positively associated with prostate cancer risk.”
A diet high in linoleic acid creates hormonal noise – excessive signaling between the cell membrane lipids and the nucleus.
This happens when linoleic acid is turned into excessive prostaglandins during times of stress, allergic reactions, and inflammation generally.
Since prostaglandins can signal the production of more prostaglandin‐forming enzymes, this can be self‐sustaining.
This can often continue until we stop eating linoleic acid or take a COX‐2 inhibitor such as aspirin.
But this is actually easy to avoid.
Most of the best foods – such as dairy, fruit, beef, and leaves – are actually quite low in linoleic acid.
In comparison, nuts, grains, and some animal products (such as chicken and pork) have considerable amounts of linoleic acid.
Our analysis suggests a positive association between prostate cancer and vertex baldness that appeared to be more evident for high-grade prostate cancer
Linoleic acid is a unique fatty acid and the only one that can be considered a prohormone.
Avoiding linoleic acid should go a long way towards avoiding cancer and hair loss.
Big Pharma makes $30 billion a year on treating diabetes. They wouldn’t make any money from a cure…
No problem… you may be able to fix diabetes yourself.
The problem is that these huge profits give incentives for Big Pharma companies to come out with all kinds of dangerous chemicals…
…instead of finding one cure that fixes diabetes for good.
I was shocked when I learned that Big Pharma doesn’t have to prove that their diabetes chemicals make you healthier or live longer.
They just have to prove that the chemical somehow lowers your blood sugar.
In other words, if it lowers your blood sugar within a year, it doesn’t matter if it kills you in 10 years…
This is how a lot of diabetes chemicals get safety-approved even though they are actually very dangerous.
And gradually, all the guys who are taking these dangerous chemicals, they get cancer or kidney failure, or they develop heart problems.
But you don’t have to buy into Big Pharma’s dangerous chemicals. You can reverse diabetes yourself, following this simple system.
Giles, Graham G. "Androgenetic alopecia and prostate cancer: findings from an Australian case-control study." Cancer Epidemiology and Prevention Biomarkers (2002)
Godley, Paul A. "Biomarkers of essential fatty acid consumption and risk of prostatic carcinoma." Cancer Epidemiology and Prevention Biomarkers (1996)
Cote, R. J. "The effect of finasteride on the prostate gland in men with elevated serum prostate-specific antigen levels." British journal of cancer (1998)
Rose, David P. "Effects of fatty acids and eicosanoid synthesis inhibitors on the growth of two human prostate cancer cell lines." The Prostate (1991)
Prostate cancer treatment is the medical treatment of individuals who suffer from any forms of Prostate cancer. When opting for surgery as a means for treating prostate cancer, the stages of the disease and the individual's overall health is usually paid attention to. The surgical options include Radical (open) prostatectomy and Robotic or laparoscopic prostatectomy. Radical prostatectomy is the surgical removal of the entire prostate and the seminal vesicles. This type of treatment has the possibility of interfering with the sexual function of the individual. In many cases it can also cause urinary incontinence, which can be normally treated with drugs or injections. The Robotic prostatectomy, as the name implies is less invasive and uses robotic instruments to do the surgery. Radiation therapy is also used in the treatment of Prostate cancer. The types of radiation therapy include External beam radiation therapy, Intensity modulated radiation therapy (IMRT), Proton therapy and Brachytherapy.The use of X-rays are modulated for the External beam and Intensity modulated radiation therapy. But in proton therapy, proton rays at high energy are used to destroy the cancer cells. Non invasive treatments such as Focal therapies for the treatment of Prostate cancer involve cry therapy or cryoablation.Cryotherapy uses extreme freezing to destroy the cancer cells ,but its propensity to cure cancer is not established yet. Systemic treatments for the treatment of prostate cancer include Androgen deprivation therapy ( ADT ) and Chemotherapy. Since the development of the prostate cancer cells is driven by the male hormones called androgens, Androgen deprivation therapy is used to slow the growth of cancer cells . The specific types of ADT are bilateral orchiectomy where the testicles are surgically removed and thus the supply of testosterone is blocked. The second approach involves taking of specific medication (LHRH agonists) which blocks the production of testosterone in the body. Chemotherapy is also used to treat prostate cancer. But with all these therapies Palliative care is very much important during the treatment phase. Palliative treatment involves the use of medication, nutrition, relaxation techniques, emotional support and other alternative methodologies to aid in the healing of the individual.
2. How fast does metastatic prostate cancer progress?
The advanced condition of Prostate cancer is called Metastatic prostate cancer and it can spread to other parts of the body. This happens in advanced cases where the cancer germs usually travel through the blood or lymphatic system. But, the progression of prostatic cancer cannot be efficiently determined since it varies from individual to individual and depends on the type of prostate cancer in any given person. Usually Prostate cancer progresses slowly in comparison to other types of cancer. The rate of progress or growth of the cancer cells usually depends on how abnormal the prostate cancer cells are. Prostate cancer cells that are high grade or composed of a high composition of abnormal cells has the propensity to spread more swiftly. The presence of other health conditions can also affect the rate at which the metastatic prostate cancer progresses. So in individuals with a weak immune system, there is a underlying propensity of a fast growth of the cancer cells. The areas where the metastatic prostate cancer progresses the most are the areas which are close to the prostate such as the seminal vesicles and lymph nodes. It also has a strong tendency to spread to the bones especially the lower spine, pelvis and the upper legs. Usually there are symptoms that signal the spread of the prostate cancer especially to the bones. There may be pain in the spine, hips and pelvis, which usually warrants the need for swift medical intervention.