Two for one? Can this simple prostate “tweak” increase rockiness but cause you to grow a full head of hair?
Why Men Go Flat (Down There)
You know how it goes…men who were roaring to go, multiple times a night, but driven beyond the bounds of nature, things stop working down there.
They are flat, they are flaccid, they are dead down there.
Why do men go flat?
Men go flat because of high estrogen…
Their T level is okay, but their estrogen is sky high. That’s what happened to me…and you know where that estrogen excess comes from?
Fat cells, that’s what creates all that extra erection-killing estrogen.
Luckily, when I found this out, I now use this simple formula to “youth-ify” my body and it melts away belly fat and MOST importantly, causes fantastic erections (due to low estrogen.)
Yes it’s a little bitter, but there is ZERO “cutting calories, counting carbs or missing meals”…and my wife loves the new “me”, LOL.
Huh? Your prostate grows hair on your head?
Many studies have demonstrated a link between prostate cancer and hair loss.
This correlation is stronger than the one between prostate cancer and cardiovascular disease…
If fact, this link has been demonstrated in all studies that have looked for it…
Except for one study that assessed hair loss based‐on frontal yearbook photos alone, with no attempt made to quantify vertex (top of head) hair loss.
On the other hand, all the truly thorough studies have shown an obvious correlation.
Finding the common denominator between hair loss and prostate cancer should help us understand the cause of both:
Following up on smaller studies which had shown a correlation, a group of Australians conducted a much larger study.
This was a study group of considerable size…
They included 1,497 cases and 1,434 control subjects.
They found a significant correlation which was also dependent on severity:
the people with the worst prostate cancer also had the most hair loss.
For high-grade disease, the ORs for all types of AA were elevated, nearly 3-fold for vertex balding
These are strong correlations that are made even more significant by the large size of the study.
This effect has been found everywhere it has been looked for…
…America, Australia, Korea, The Netherlands, and France.
Conducted in Europe with 669 subjects, this study found the same correlation.
And these subjects were young, all under 20.
This all but rules out most of the possible confounders. (A confounder is an unobserved exposure that could affect outcomes.)
The fact that this correlation is observable across ages, and across geographic locations, suggests that it represent a very real effect.
When compared with control group subjects, those with prostate cancer were twice as likely to have signs of alopecia at age 20
So how do they explain it?
Well, they just mumble something about androgens or course…
Even though testosterone has actually been shown to stimulate hair growth in vitro…
And finasteride doesn’t slow prostate cancer – at all.
In fact, the only significant finding from the use of finasteride in the study below was actually an increase in prostate cancer:
Caught up in the prostate cancer–androgen paradigm, these researchers decided to conduct a study into just that…
Men with prostate cancer, and some without, were given finasteride for one year.
The results were measured by biopsy, and then compared with one taken a year before.
The slides were coded (meaning it was a blind study, removing bias from the results).
Moreover, in patients with pre-existing PIN lesions, treatment with finasteride was statistically significantly associated with an increased incidence of prostate cancer at 1 year.
So there you go, an increase in prostate cancer from finasteride.
If androgens were responsible for prostate cancer, then finasteride should be the wonder drug.
But it’s not. Finasteride had the opposite effect in this study.
It’s true that lowering androgens can lead to a decrease in prostate volume, since androgens are always anabolic and the prostate is an androgen‐dependent organ.
But this fails to explain cancer – since volume is not cancer.
Cancer is cancer. And smaller people – with less volume – still get cancer.
If the androgens were truly the cause…
There would be no need for terms such as ‘‘androgen‐refractory prostate cancer”.
That phrase tacitly admits that androgens are not a primary factor, while also implying how people just assume they ought to be.
Pinning every sex difference on androgens is just lazy, and women don’t even have prostates.
This study raises serious questions about the probable efficacy of finasteride in preventing prostate cancer.
Androgens cannot explain the geographic incidence either.
Men everywhere have essentially the same amount of androgens, yet vary wildly in prostate cancer rates.
Generally, you see a trend the further north you go.
Genetics can’t explain this. Androgens can’t explain this.
These geographic patterns are not readily explicable by other known risk factors for prostate cancer.
More realistic risk factors probably involve vitamin D…
Or perhaps a dietary factor such as linoleic acid.
Plants synthesize more linoleic acid the further from the equator they are.
This is probably because unsaturated fatty acids have a low freezing point, helping the plants resist colder climates.
Northerners eat more grains because grains are suitable to be grown northern climates.
While it’s true that we could all probably use a bit more vitamin D…
Prostaglandins derive solely from one dietary source – linoleic acid.
Prostaglandin D₂ is nearly synonymous with hair loss (Garza, 2012), and linoleic acid has been shown to accelerate prostate cancer in vitro (Rose, 1991.)
And linoleic acid has been found to correlate directly with prostate cancer, to a very high degree:
In this classic study, Paul Godley compared prostate cancer to individual fatty acids.
This wasn’t just a flaky dietary recall study, his team actually took biopsies of adipose tissue.
Animal studies have suggested that omega−6 fatty acids found in vegetable oils may promote prostate cancer.
They determined which fatty acids were present within the body and in what ratios.
Then they correlated these with prostate cancer…
They found unbelievably high risk ratios.
The observed correlations we so strong in fact, that you’ll forget all about androgens.
This was observed exclusively with one fatty acid: linoleic acid…
Which happens to be the only fatty acid that acts as a precursor to prostaglandins.
In Table 4, only linoleic acid demonstrated a statistically significant increase in risk
Consider also the in vitro demonstrations by Rose, who observed cancer cell growth with linoleic acid…
And you’ve essentially found the #1 risk factor for prostate cancer.
Luis Garza, among others, has shown that prostaglandin D₂ is highly correlated with hair loss, in every way.
The common denominator appears to be linoleic acid…
And linoleic acid is what prostaglandins are made of.
a possible relationship between the rising breast cancer risk and an increase in the consumption of vegetable oils rich in linoleic acid (found in vegetable oil). This may also apply to prostate cancer.
Considering the research on prostaglandins alone, we could actually expect hair loss would be correlated with prostate cancer, just as observed.
Prostaglandins can be considered hormones.
They are lipid signallers which tell a cell that it’s under attack.
These signal the receptors in the nucleus, telling them to create more fat-metabolizing enzymes to help get rid of excessive prostaglandins.
But this shifts metabolism away from natural metabolism, and towards a pathological state of affairs.
Obviously, before we knew how to make fire, humans mostly lived in the tropics – where linoleic acid is scarce.
And the few exceptions who lived in the North, such as the Neanderthals…
…probably had enough sense not to use canola oil.
Linoleic acid consumption (in vegetable oil) was positively associated with prostate cancer risk.
A diet high in linoleic acid creates hormonal noise – excessive signalling between the cell membrane lipids and the nucleus.
This happens when linoleic acid is turned into excessive prostaglandins during stress, allergic reactions, and inflammation generally.
Since prostaglandins can signal the production of more prostaglandin‐forming enzymes, this can be self‐sustaining.
This can often continue until we stop eating linoleic acid or take a COX‐2 inhibitor such as aspirin.
But luckily, this is actually easy to avoid.
Most of the best foods – such as dairy, fruit, beef, and leaves – are actually quite low in linoleic acid.
In comparison, nuts, grains, and some animal products (such as chicken and pork) have considerable amounts of linoleic acid.
Our analysis suggests a positive association between prostate cancer and vertex baldness that appeared to be more evident for high-grade prostate cancer
Linoleic acid is the most unique fatty acid, and the only one which can be considered a prohormone.
Avoiding linoleic acid should go a long way towards avoiding cancer and hair loss.
Big Pharma makes $30 billion a year on treating diabetes. They wouldn’t make any money from a cure…
No problem – you may be able to fix diabetes yourself.
The problem is that these huge profits give incentives for Big Pharma companies to come out with all kinds of dangerous chemicals…
…instead of finding one cure that fixes diabetes for good.
I was shocked when I learned that Big Pharma doesn’t have to prove that their diabetes chemicals make you live longer or healthier.
They just have to prove that the chemical somehow lowers your blood sugar.
In other words, if it lowers your blood sugar within a year, it doesn’t matter if it kills you in 10 years…
This is how a lot of diabetes chemicals get safety-approved even though they are actually very dangerous.
And gradually, all these guys taking these dangerous chemicals, they get cancer, or kidney failure, or develop heart problems.
But you don’t have to subscribe to Big Pharma’s dangerous chemicals – you can reverse diabetes yourself, following this simple system.
Giles, Graham G. "Androgenetic alopecia and prostate cancer: findings from an Australian case-control study." Cancer Epidemiology and Prevention Biomarkers (2002)
Godley, Paul A. "Biomarkers of essential fatty acid consumption and risk of prostatic carcinoma." Cancer Epidemiology and Prevention Biomarkers (1996)
Cote, R. J. "The effect of finasteride on the prostate gland in men with elevated serum prostate-specific antigen levels." British journal of cancer (1998)
Rose, David P. "Effects of fatty acids and eicosanoid synthesis inhibitors on the growth of two human prostate cancer cell lines." The Prostate (1991)