Most important male hormone is not testosterone

Everyone thinks that testosterone is the most important male androgen…

…but it’s actually a different hormone that doctors never talk about…

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—-Important Message—-

This forgotten natural hormone works 113 times better than testosterone for men’s erections and lasting power

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The next time you want to boost your erections or bedroom performance — skip the testosterone supplements…

Instead, try this forgotten natural hormone I’ve discovered that is even better for erections than testosterone.

This is super good news for men who think their testosterone is low…

Now you can get the best erections of your life without worrying about your T levels!

Because as I’ve discovered, testosterone isn’t the end all, be all when it comes to getting great erections as a man…

…it’s this forgotten natural hormone that works 113 times better than testosterone

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Is serotonin really the happy hormone?

Serotonin has been heavily marketed as a beneficial “happy hormone” since the 1980s.

This claim is absurd – as I have explained in many articles and some newsletters.

Excess serotonin causes anxiety, depression, aggression, fatigue, gut problems and metabolic issues like type II diabetes.

Serotonin can be useful if you’ve eaten something toxic – because serotonin triggers vomiting and diarrhea.

But the reality is that most people are suffering with too much serotonin – not too little.

Serotonin is created by an enzyme called TPH

And researchers have discovered that a youth-associated hormone called DHEA

…down-regulated stress-related increases in TPH in the brain.

A little DHEA can help to lower one of the most problematic stress hormones, serotonin…

And help with mood disorders, gut problems, and metabolic issues.

DHEA blocks increases in brain serotonin via TPH

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The animal experiments were performed at Virginia Commonwealth University in Richmond. The paper was published in Molecular and Cellular Neuroscience.

The authors of the study were carrying out experiments looking at the biochemical effects of stress on rodents.

In these experiments they were using loud noise to stress out rats.

They showed that stress increases TPH in the brains of the animals.

TPH is the enzyme which synthesizes serotonin.

But when they injected the animals with DHEA they found that this prevented the stress related increase in serotonin…

By preventing the increase in TPH.

“Infusion of DHEA blocked the sound stress-induced increase in TPH activity observed ex vivo in midbrain and cortex but had no effect on the level of TPH activity from sham-stressed rats.”

DHEA is a hormone produced in the body.

Young people produce lots of it – both men and women.

The levels of DHEA tend to decrease as people get older.

Numerous human and animal experiments have shown that DHEA improves the immune system and slows aging.

It also has potent anti stress effects — and its ability to lower TPH may explain some of those effects.

The experiments showed that DHEA injections into varying different parts of the brain were effective at lowering TPH and stress-related serotonin.

“DHEA (20 micrograms total dose) given bilaterally into the region of the central nucleus of the amygdala, 30 min prior to 1 h sound stress, also blocked the increase in TPH in a dose-dependent manner.”

Another interesting finding was that other related hormones did not have the same effect as DHEA.

“The effect of DHEA was steroid specific in that other sex steroids, such as estrogen, androgens, or progesterone, were without any effect.”

Later on, the researchers found that the mechanism of action may have something to do with glucocorticoid receptors.

These are the receptors which, in humans, are activated by another primary stress hormone – cortisol.

“Coadministration, 20 micrograms each, of a potent glucocorticoid blocker with DHEA 30 min prior to ound stress completely blocked the DHEA suppressive effect on sound stress-induced increases in TPH activity.”

The anti-cortisol effects of DHEA were known long before this research was carried out.

“The results obtained suggest that DHEA blocks this increase in TPH activity by antagonizing the effects of glucocorticoid.”

The study shows that DHEA has a remarkable effect at blocking stress-related increases in serotonin…

As well as its already known anti-cortisol effect.

DHEA is a remarkable antistress substance.

And it can be beneficial for people to increase in DHEA – indirectly by diet and lifestyle hacks or by taking DHEA itself.

The caveat is that you must not take too much. 

Excess DHEA will convert to estrogen – the other primary stress hormone.

Topical applications of 3 to 5mg DHEA up to 3 times per day are effective at lowering stress without increasing estrogen.

—-Important Message—-

Think you need DHEA or testosterone? Maybe not…

Once a man begins taking DHEA or testosterone supplements from the doc, his body stops producing as much on its own.

Plus, the DHEA or testosterone he’s taking eventually gets turned into estrogen…

Not good for a man.

So now, the man needs more and more fake testosterone, but that just keeps getting turned into estrogen…

…and soon it all becomes a downward spiral for the man’s libido and performance.

So now, knowing all this…

…the only thing left is to figure out how to naturally raise testosterone while keeping estrogen low…

———-


Matt Cook is editor-in-chief of Daily Medical Discoveries. Matt has been a full time health researcher for 26 years. ABC News interviewed Matt on sexual health issues not long ago. Matt is widely quoted on over 1,000,000 websites. He has over 300,000 daily newsletter readers. Daily Medical Discoveries finds hidden, buried or ignored medical studies through the lens of 100 years of proven science. Matt heads up the editorial team of scientists and health researchers. Each discovery is based upon primary studies from peer reviewed science sources following the Daily Medical Discoveries 7 Step Process to ensure accuracy.
Intracranial Dehydroepiandrosterone blocks the activation of TPH in response to acute sound stresshttps://pubmed.ncbi.nlm.nih.gov/8032685/