Lisa confesses her sickening sex secret

Now this is the only thing that will make her feel better…

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—-Special Message From Lisa—-

“…thank God we tried THIS first…”

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My husband suddenly had zero desire for me…

His drive was low, his confidence was low, and as I told my girlfriend (she is sworn to secrecy)…his penis wasn’t really doing it for me…

…and I had no idea how to help him…

Things got so bad, I almost made a HUGE mistake that would have cost me my marriage…

Luckily, I discovered these testosterone-boosting life-savers in the nick of time…

Just one bite and my husband was back to the man I married: sexy, confident, and back to blowing my mind in bed!

———-

Here’s why this is my favorite over the counter treatment

Aspirin is one of the oldest and still one of the most useful treatments.

Many people take low doses of aspirin to prevent cardiovascular diseases or lower the risk of cancers – and with good reason.

The list of benefits of aspirin is almost endless – and yet there are still beneficial mechanisms to aspirin which are just being discovered.

Recently, researchers found that aspirin decreases cardiovascular inflammation and portal hypertension.

The mechanism is related to angiotensin II – the target for ACE inhibitor treatments.

This science uncovers yet another mechanism by which aspirin can protect the cardiovascular system…

…and also act against more general inflammatory conditions.

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The animal experiments were carried out at the Humboldt University of Berlin. The paper was published in FASEB Journal.

Angiotensin II is produced in the body.

It increases blood pressure and causes the kidneys to retain sodium and water.

It also stimulates the release of a hormone called aldosterone, which is harmful in excess.

Angiotensin II causes inflammation of the walls of the blood vessels.

It does this by activating an inflammatory protein called NF-κB.

“Angiotensin II induces vascular wall inflammation by activating IKKβ.”

IKKβ is a protein which sits between angiotensin II and NF-κB.

Angiotensin II activates IKKβ.

IKKβ activates NF-κB.

NF-κB causes inflammation of the blood vessel walls which lead to cardiovascular disease and death.

Aspirin lowers IKKβ in the lab (in vitro).

If it does the same in live animals (in vivo) it could provide a mechanism to lower inflammation and decrease cardiovascular problems.

“Aspirin inhibits IKKβ in vitro – however, the in vivo relevance of the phenomenon is unclear.”

The researchers were curious whether aspirin could decrease inflammation of the walls of the blood vessel by inhibiting IKKβ…

Thereby lowering inflammatory NF-κB.

“We tested the hypothesis that aspirin protects from Ang II-induced end organ damage by inhibiting NF-κB activation in vivo.”

They carried out their experiments on rats which had been modified to have the relevant human angiotensin genes.

Rats were given low or high doses of aspirin to see the effects.

“Rats harboring human renin and angiotensinogen genes received high-dose or low-dose aspirin.”

High-dose aspirin had a remarkable protective effect on the heart and kidneys.

Fewer rats died from cardiovascular inflammation when given high-dose aspirin.

The researchers also found aspirin prevented structural changes to the heart and protected the kidneys.

“High-dose aspirin reduced mortality, cardiac hypertrophy, fibrosis, and albuminuria independent of blood pressure”

The researchers believe that their theory is correct – that aspirin protects blood vessel walls against inflammation by lowering NF-κB.

(Aspirin directly lowers IKKβ, which in turn decreases the inflammatory NF-κB.)

“High-dose aspirin inhibits NF-κB activation and inflammation in heart and kidney.”

The study shows yet another beneficial effect of aspirin on the cardiovascular system.

It goes some way to explaining why people who take smart amounts of aspirin…

…are significantly less likely to suffer from major cardiovascular diseases and death.

But NF-κB-related inflammation does not just affect the cardiovascular system.

This research shows that it also affects the kidney.

The truth is that the inflammatory and disease-causing effects of NF-κB can be found right throughout the body.

It is an almost ubiquitous promoter of inflammation.

And so this research also shows that aspirin can have beneficial effects on all sorts of inflammatory diseases.

Aspirin works by its effect on the inflammatory proteins discussed already in this article.

“These in vivo results serve to explain the clinical utility of high-dose aspirin in inflammatory disorders and suggest additional therapeutic avenues that may be relevant to cardiovascular disease.”

Low-dose aspirin is safe for almost everybody…

Though you do need to be careful if you are taking any type of blood thinner as there can be potent interactions.

—-Important Message About Avoiding Heart Attack—-

The nitric oxide hoax — what all men need to know

Unfortunately, I’ve heard from thousands of men who have suffered heart attacks…

…and they all had one thing in common.

They were all taking heart meds and cholesterol-lowering treatments that were supposed to keep that from happening.

Turns out, those treatments can make heart problems WORSE.

Unfortunately, many men have to discover this the hard way… but you don’t have to…

Discover a natural solution some are calling Nature’s Artery Opener and save your heart while you still can.

———-


Matt Cook is editor-in-chief of Daily Medical Discoveries. Matt has been a full time health researcher for 26 years. ABC News interviewed Matt on sexual health issues not long ago. Matt is widely quoted on over 1,000,000 websites. He has over 300,000 daily newsletter readers. Daily Medical Discoveries finds hidden, buried or ignored medical studies through the lens of 100 years of proven science. Matt heads up the editorial team of scientists and health researchers. Each discovery is based upon primary studies from peer reviewed science sources following the Daily Medical Discoveries 7 Step Process to ensure accuracy.
Aspirin inhibits NF-κB and protects from angiotensin II-induced organ damagehttps://faseb.onlinelibrary.wiley.com/doi/10.1096/fj.00-0843fje