Scientists are saying this 1 over the counter treatment is slowing down mitosis
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How to slow down mitosis and stop cancer
Colorectal cancer is the 5th most common form of cancer.
One of the problems in cancer is that cells multiply too rapidly, forming tumors.
Mitosis is the process by which one cell divides into 2 cells.
It can be a normal and healthy process by which cells are replenished in the body.
Mitosis is cell division and multiplication.
But when mitosis becomes too rapid, it plays into the development of cancer.
It is one of the main mechanisms of colorectal cancer.
Researchers have recently discovered that aspirin slows down the process of mitosis.
This means that rapidly dividing cells are more likely to be kept under control.
This could explain one reason why aspirin lowers the risk of this disease.
The human research was carried out at Emory University School of Medicine in Atlanta. The paper was published in BMC BioMed Central.
Epigenetics is the term given to factors which influence genes.
This study was interested in the effect of aspirin on epigenetic factors relating to out-of-control cell division in colorectal cancer.
“Despite the known role of mitosis in colorectal cancer, previous associations of long-term aspirin use with suppressed cancer-related epigenetic aging did not involve epigenetic mitotic clocks.”
Mitotic clocks are processes which affect how rapidly cells divide and multiply.
Slowing mitotic clocks can make cells divide and multiply more slowly – decreasing cancer risk.
“We investigated these relationships using three epigenetic mitotic clocks developed for cancer risk prediction.”
The researchers carried out their experiments using over 100 samples taken from different parts of the colons of a couple of dozen people.
“We utilized publicly available data from 112 healthy colon (proximal and distal) mucosal samples taken at baseline and at 10-years follow-up.”
Some of the participants had been taking aspirin for a long period of time (more than 2 years).
The remaining participants were not aspirin users.
“Our samples were taken from a screening cohort of 28 Polish women (11 non-users and 17 long-term aspirin users).”
The researchers looked at the epigenetic factors which affect the rate of cell division (mitosis) in all of the samples.
They then factored in the age of the people who the samples were taken from – something which is known to affect the rate of mitosis.
“Mitotic clock values were divided by chronological age at each timepoint to obtain intrinsic rates (IRs).”
The intrinsic rate is the rate of cell division and multiplication (mitosis) after accounting for other things like age.
Other important factors like whether or not the participants had polyps and their BMI were also factored in.
The researchers found a significant decrease in mitosis in the colon in long-term aspirin users.
It seems that aspirin was slowing down the rate of cell division and multiplication in the colons of these individuals.
“We observed deceleration for all 3 mitotic clocks for long-term aspirin users from time 1 to time 2 (10 years later).”
The same slowing of cell division and multiplication was not seen in people who were not taking aspirin.
“This slowing of mitotic cancer related clocks was not found in aspirin non-users.”
The research provides a solid mechanism by which aspirin can be said to decrease the risk of cancer.
50 years ago, many people thought that cancer was hardwired by genes.
But we now know that epigenetic factors can influence genes and change outcomes.
It seems that one of these epigenetic factors in colorectal cancer is aspirin.
Previous studies have shown that regular use of aspirin seems to reduce the risk of colorectal cancer by 25-30%.
Aspirin seems to prevent the spread of colorectal cancer to the liver.
We also know that aspirin has an effect on gut bacteria which is likely to lower the chances of colorectal cancer too.
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