Erase 20 years of old age with this 1 food?

Gurus say to avoid it — but men living to 100 are eating it…

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—-Important Message—-

Erase 20 years of old age with this 1 food?

I know it sounds crazy, but there’s 1 food that very old and vital men are eating — and they’re often living to 100, 110 or even 120…

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While many so-called health gurus are dropping dead at 50 or 60 by AVOIDING this one food…

Here’s the 1 food — are you eating it or are you avoiding it?

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Drinking this daily can increase your lifespan by decades

Humans have always wanted to increase longevity.

Who wouldn’t want to live longer – provided you lived in decent health?

Studies in microorganisms and small animals have shown that dietary restriction can increase longevity.

Later research showed that much of the longevity increase was achieved by simply decreasing an amino acid called methionine.

So, fasting and caloric restriction are probably unnecessary.

We also know that supplementing glycine helps the body to remove excess methionine.

Glycine significantly increases the lifespan of small animals.

Rapamycin is a treatment which is used primarily to prevent organ transplant rejection.

And it’s also a promising candidate for treatment-induced longevity.

Researchers have recently discovered that some of the biological effects of caffeine are similar to that of rapamycin.

What is more, caffeine increases the lifespan of microorganisms – and it comes without many of the risks associated with rapamycin.

The findings may partly explain why coffee drinkers tend to live longer.

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The microorganism research was carried out at the University of Geneva Medical School in Switzerland. The paper was published in Molecular Microbiology.

The authors of this study were interested in the effect of caloric restriction on longevity.

Restricting calories, or restricting protein, or restricting methionine leads to extended life in small organisms.

“Dietary nutrient limitation (dietary restriction) is known to increase lifespan in a variety of organisms.”

The authors were working on a puzzle that has already been solved. (It’s methionine restriction, which increases longevity.)

Nevertheless, the research did uncover some useful findings.

They carried out their experiments on yeast.

The researchers looked at the effect of different substances on proteins known to be related to longevity.

These include TORC1 and Sch9.

“Although the molecular events that couple dietary restriction to increased lifespan are not clear, studies of the model eukaryote Saccharomyces cerevisiae have implicated several nutrient-sensitive kinases, including the target of rapamycin complex 1 (TORC1) and Sch9.”

In the course of their experiments they found that caffeine acts on these proteins in a way similar to rapamycin…

…a well-publicized treatment which increases longevity in microorganisms.

Caffeine or rapamycin have similar knock-on effects on these proteins – and they believe that these mechanisms may lead to increased lifespan.

One of these proteins (TORC1) is also involved in sensing how much methionine is consumed in the diet.

Previous studies have shown that excessive methionine shortens lifespan and decreasing methionine helps animals to live longer.

Other investigations into longevity-promoting substances have shown that aspirin is perhaps the most potent life extender.

Both aspirin and caffeine are very effective at increasing the metabolic rate.

This helps the body to produce more energy, which can be used for repair and recovery – this surely plays into increased longevity too.

Whatever the exact combination of mechanisms may be…

…the researchers showed in their experiments that caffeine increases longevity.

“Treatment of yeast cells with the specific TORC1 inhibitor rapamycin or caffeine releases Rim15 from TORC1-Sch9-mediated inhibition and consequently increases lifespan.”

Although the researchers were working on yeast, the proteins and enzymes in question are also present in humans and act in much the same way as they do in yeast.

The authors of the study concluded that caffeine may therefore increase longevity.

And there is already evidence to show that consuming caffeine (in coffee) could increase human longevity.

Coffee decreases the risk of death from most of the major killers.

“This cascade appears to have been evolutionarily conserved, suggesting that caffeine may extend lifespan in man.

You should always consult your healthcare practitioner for guidance on medical diagnosis and treatment.

—-Important Message About Deadly Blood Fat—-

Blood fat is clogging up men’s penile chambers and hurting erections — so drink THIS to clear the penile chambers and destroy blood fat for good

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Picture a plastic straw and then fill it up with wax.

Now imagine trying to use it while it’s clogged up like that…

How much liquid do you think you’re going to get?

Well, this clogged up straw is a man’s member when his penile chambers are blocked up…

And they get blocked up by something called blood fat… a terrible, awful toxin that hurts the male body in so many ways…

So it’s no wonder men aren’t getting good erections when they have blood fat blocking off blood flow down there.

It’s blood fat to blame…but doctors don’t know about blood fat, so most guys don’t either!

Luckily, there’s an easy fix — this washes the penile chambers clean from blood fat for better erections than you can ever remember 

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Matt Cook is editor-in-chief of Daily Medical Discoveries. Matt has been a full time health researcher for 26 years. ABC News interviewed Matt on sexual health issues not long ago. Matt is widely quoted on over 1,000,000 websites. He has over 300,000 daily newsletter readers. Daily Medical Discoveries finds hidden, buried or ignored medical studies through the lens of 100 years of proven science. Matt heads up the editorial team of scientists and health researchers. Each discovery is based upon primary studies from peer reviewed science sources following the Daily Medical Discoveries 7 Step Process to ensure accuracy.
Caffeine extends yeast lifespan by targeting TORC1https://pubmed.ncbi.nlm.nih.gov/18513215/