Is This Cancer Treatment is Being Ignored and Suppressed?

Is This Cancer Treatment is Being Ignored and Suppressed?

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Have you ever wondered if they’ll ever figure out how to cure cancer?

Some people think the cure was found, but that the information is suppressed — for nearly a century.

Why would anyone suppress cancer cures?  Hmmm….

Our story starts in the 1920’s.  

Detroit physician Dr. William Koch claimed to have discovered a compound that could reverse cancer.

He wrote in later books that the AMA harassed him and that they barred him from publishing his findings.

However, any further attempts to publish any subsequent re­ports were blocked, at the request of the Journal of the A.M.A., so that the official medical journals refused me the right to report my findings.

Decades later, Nobel Prize winning biochemist Albert Szent-Györgi independently discovered the same compound.

And the remarked extensively on its anticancer effects.

Szent-Györgi was not a medical doctor, so he only worked with animals.

Nonetheless, he has some remarkable things to say about it:

I have shown that MG, at a low concentration, can arrest cell division reversibly, without harming the cell, and so could play a central role in cellular regulation.

The compound is methylglyoxal (MG), and it occurs naturally in normal cells.

This research happened during the ‘70s while Richard Nixon’s “War on Cancer” was a big deal.

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His “War” convinced the public mind that a cure for cancer remained elusive.

But according to these two famous biologists, the cure is already proven — TWICE.

And it doesn’t end there, derivatives of this drug are being used clinically in Europe and Mexico.

Plus, another molecule called 3-bromopyruvate showed great results recently in reversing tumors.

This molecule is remarkably similar to Koch’s and Szent-Györgi’s methylglyoxal.

It’s small and has the type of chemical structure that Szent-Györgi claimed safely inhibited cell division.

We call the structure dicarbonyl, and it’s a common chemical group that has two carbon atoms.

I’m not sure why that’s important, but it is.

The reason could be because cancer breaks down the electron transport chain in cells.  

The electron transport chain is part of the cell’s metabolism. 

And these drugs work by correcting this metabolism issue.

A team of researchers from Johns Hopkins University looked at the effect of the 3-bromopyruvate compound.

They found a striking illustration of its cancer-reversing effect.

In the study, researchers anesthetized mice and injected them with cancer cells.

Then they treated one group with 3-bromopyruvate, and another with only a placebo.

The team achieved a 100% remission rate in the 19 mice that received 3-bromopyruvate.

All advanced solid tumors growing in the upper backs of test animals also regressed and disappeared following treatment with 3-BrPA.

And all of the saline-injected placebo rats died — that’s a clear picture of how effective the 3-BrPA really is.

It’s not possible to have a more effective drug than this.

The researchers took color photographs since words can’t adequately do justice to the tumor-reversing effects of bromopyruvate.

It COMPLETELY reversed the tumors in all 19 mice.

And bromopyruvate does not seem to affect normal tissue.

In fact, it appears that only cancerous cells take it in.  

Nearly all cancer treatments we use hurt healthy cells, so this is an incredible finding.

A treatment that hurts cancer, but leaves healthy cells completely intact is almost miraculous.

And we have another study today that shows how the 3-BrPA cancer treatment works this miracle.

Different researchers conducted this study.

And we show distribution data below on how cancerous cells concentrate this drug:

This table shows a 15x greater drug concentration in the cancer cells than in blood.

Which tells us that tumor cells have a higher affinity for 3-bromopyruvate than healthy cells do.

This second study used rabbits, and all of the rabbits lived much longer in the 3-BrPA group.

The mean survival time in the control group was 18.6 days, whereas the mean survival time in the treatment group was 55.0 days, showing a statistically significant 296% increase in survival for the rabbits treated i.a. (intra-arterially) with 3-BrPA.

A 296% increase in survival from only ONE 3-BrPA infusion.

And 3-bromopyruvate is much less toxic than most standard chemotherapy drugs.

Noteworthy is the absence of toxicity to the normal liver on histopathological analysis, which is in keeping with previous data from our laboratory.

This is what William Koch was doing in the ‘20s.

He was achieving high cure rates in people, nearly 100 years ago!

Still, William Koch lost his funding, endured two AMA court cases, and fled to Brazil.

But these court cases unveiled more proof of the efficacy of methylglyoxal.

He was exonerated in both trials because so many people testified on his behalf that the drug actually worked.

And yet this treatment is still not available in the United States.

Makes you wonder why doesn’t it?

 


Matt Cook is editor-in-chief of Daily Medical Discoveries. Matt has been a full time health researcher for 26 years. ABC News interviewed Matt on sexual health issues not long ago. Matt is widely quoted on over 1,000,000 websites. He has over 300,000 daily newsletter readers. Daily Medical Discoveries finds hidden, buried or ignored medical studies through the lens of 100 years of proven science. Matt heads up the editorial team of scientists and health researchers. Each discovery is based upon primary studies from peer reviewed science sources following the Daily Medical Discoveries 7 Step Process to ensure accuracy.
Advanced cancers: Eradication in all cases using 3-bromopyrivate therapy to deplete ATP
https://www.researchgate.net/publication/8250635_Advanced_cancers_Eradication_in_all_cases_using_3-bromopyruvate_therapy_to_deplete_ATP 

Targeting of VX2 Rabbit Liver Tumor by Selective Delivery of 3-Bromopyruvate: A Biodistribution and Survival Study 
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2760588/ 

The Living State and Cancer 
http://www.pnas.org/content/74/7/2844.full.pdf 

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