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Yes, nearly twice as effective as testosterone in improving your erections, sexual performance, and stamina.
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How these five blood pressure treatments cause sexual dysfunction
If you are taking any of these 5 blood pressure treatments, you may need to talk to your doctor:
Diuretics may decrease the body’s levels of the mineral potassium, which can lead to erection problems in some men. Also weakness, leg cramps, or fatigue.
Beta-blockers make your heart beat less forcefully and more slowly. This may cause asthma symptoms, cold hands and feet, depression, and erection problems.
ACE inhibitors are notorious for causing erection problems for virtually all men taking them. Also, they may cause a dry, hacking cough that doesn’t go away. Skin rash and a loss of taste are two other possible side effects of ACE inhibitors.
Alpha-blockers reduce nerve impulses. They affect blood vessels, but they also affect every cell in the body. This may cause dizziness, lightheadedness, or weakness when standing up suddenly or getting up in the morning (due to reduced blood pressure).
Vasodilators relax muscles in blood vessel walls. That may cause fluid retention, headaches, irregular or very rapid heartbeat (palpitations), joint aches and pains, swelling around the eyes.
As you can see, all the Big Pharma solutions just make things worse for men…
But I discovered a simple, natural way to lower blood pressure and restore erections if you’ve been taking any of those nasty Big Pharma chemicals.
Here’s the natural way to lower blood pressure and experience great erections again…
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Better than testosterone?
Scientists have known for decades that androgens regulate nerve growth.
In males, androgens are essential during development to masculinize the nervous system – leading to changes in nerve patterns in the lower spine and brain.
They have also been shown to greatly stimulate neurite outgrowth in vitro.
But when this first became known, decades ago, scientists assumed that testosterone itself was responsible for these effects.
It had even been hypothesized – strangely – that it did so by becoming estrogen first, through the enzyme aromatase.
Subsequent studies revealed that it was, in fact, DHT that was most active.
Testosterone becomes DHT through the enzyme 5α-reductase.
In fact, it has been known for even longer that DHT is a stronger androgen than testosterone itself…
That is to say, it has a higher affinity for the androgen receptor.
This study (among others) effectively illustrates this.
The scientists were designing potent antiandrogens in this study.
They provide data on the binding affinity for the androgen receptor of both testosterone and DHT.
DHT binds almost twice as strongly as testosterone.
That means it’s nearly twice as effective (over testosterone) as an androgen.
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The only “androgens” stronger than DHT are synthetic androgens.
So DHT is, in fact, the most potent natural androgen.
According to Wikipedia’s article on dihydrotestosterone:
“Relative to testosterone, DHT is considerably more potent as an agonist of the androgen receptor (AR).”
You occasionally hear loose talk about “weak androgens,” which refers to steroids such as DHEA and androstenedione.
Nobody, besides the pharma company Merck and a few medical doctors, have the audacity to imply otherwise.
How many people would take finasteride if they knew that DHT was the primary androgen?
And how many people would take finasteride after reading this article?
This article plainly shows that DHT, not testosterone itself, is responsible for the neurogenic effects of administering testosterone.
Since testosterone turns into DHT in the body, it takes a considerable effort to determine which actions can be ascribed to which hormone.
These endocrinologists did just that…
They studied the effects of testosterone, DHT, and finasteride on nerve cells.
They studied the effects of the two androgens in expressing mRNA (messenger RNA) for neuritin.
Neuritin is one of the most powerful nerve growth factors there is.
As you can see from figure 3a, just 1 nM of DHT was as effective as 100 nM of testosterone – another illustration of DHT being the strongest androgen.
And finasteride completely inhibited this effect – it blocked all new neurite growth.
“In fact, finasteride, a selective 5α-R 2 inhibitor that blocks DHT formation, completely counteracted the trophic effect exerted by testosterone on neurite outgrowth.”
(5α-R 2 inhibitor is a DHT blocker.)
The effects of DHT on motor neurons are best illustrated through development.
Androgens cause changes in the male nervous system in certain areas.
They actually stimulate growth in the spinal nucleus of the bulbocavernosus, which creates the motor neurons necessary for sex.
And since DHT is the most powerful androgen, you would expect it to have the greatest effect.
This can go a long way to explain the sexual side effects experienced by finasteride users…
…since these androgen-dependent nerves are essential for normal activity.
“The effects of testosterone appear to be mediated by its 5α-reduced derivative [DHT].”
Many other studies show the same nerve-promoting effect…
Androgens – especially DHT – promote new motor nerve growth and increase survivability in brain cells exposed to a toxic insult.
Below is one such study:
In this study, Dr. Ramsden used a powerful neurotoxin – kainate – to cause cell death in rat brains.
The study used three primary groups of rats: a control group, a castrated group, and a castrated group given DHT (largely compensating for the androgen-producing organs removed by castration).
The rats using DHT were mostly protected from the kainate neurotoxin.
After injection, they retained more neurons in their brains.
The implantation of a slow-release DHT pellet left the androgen group with 176% more neurons than the non-DHT castrated group.
“DHT replacement in GDX male rats significantly reduced the severity of lesion-induced neuronal loss…”
Nerve cells have an androgen receptor.
And Schwann cells – the cells that help produce the myelin sheath – have a progesterone receptor.
It was shown by Magnaghi in 1999 that DHT – and not testosterone – can activate the progesterone receptor.
They also found that myelin protein zero (P0) is upregulated by DHT and not by testosterone.
This protein makes up over 50% of the total protein of the myelin sheath.
So this protein basically is myelin – along with pregnenolone, progesterone, and DHEA.
Okay, in this study they injected castrated rats with either testosterone or DHT – and killed them 24 hours later for testing.
They also did a similar experiment with isolated Schwann cells, the myelin-producing cells.
After analyzing how much myelin protein (P0) was produced – and how much mRNA was produced to encode for that protein – they came to the conclusion that:
“DHT is able to exert, at least at the 100 and 1,000 nM concentrations, a transcriptional activity in this system via the human progesterone receptor. In contrast, testosterone did not display any transcriptional activity at any doses utilized.”
Based on the evidence, this was a reasonable conclusion.
At 1 μM:
- Progesterone induced transcription of 187×
- DHT of 79×
- Testosterone of less than 2×
So besides being a much stronger androgen than testosterone, DHT has other functions:
It can increase myelin production through the progesterone receptor.
Reduced DHT is not the only thing to worry about while taking finasteride.
The enzyme 5α-reductase is necessary for converting progesterone into 5α-progesterone – an important steroid in its own right.
“The lack of effect of testosterone in the ‘in vivo’ experiments is not surprising since testosterone has a much lower affinity for AR than DHT…”
The isolated in vitro Schwann cells produced much more myelin protein (P0) in response to DHT.
The same results were seen with injections, although in this case DHT’s superiority over testosterone was slightly diminished since testosterone becomes DHT.
So finasteride might be expected to reduce neurite growth and myelination – especially in the characteristically male regions.
“Orchidectomy [castration] has been shown to bring about a significant decrease of P0 mRNA [myelin protein zero mRNA] in the sciatic nerve of adult male rats – and this effect is counteracted ‘in vivo’ by treatment with DHT.”
So now we have considerable scientific proof…
Most of the effects on the nervous system previously attributed to testosterone are actually the result of DHT – the main metabolite of testosterone.
The mass media – and the advertisers that support them – give the impression that DHT is a “minor” androgen…
AND that inhibiting DHT with expensive drugs is a good idea.
Many people now realize that’s a terrible idea…
But the drug patents are still in force and such drugs will continue to be sold to those willing to buy them.
AND there is actually more evidence that cortisol is more of a factor in hair loss than DHT.
—-Important Message—-
How to use DHT to get 30-minute erections
I have a friend who recently went to a clinic for testosterone-replacement therapy…
He was so excited to have high testosterone again and he couldn’t wait to experience sexual bliss in the bedroom!
But after a few days, he got very tired and woozy… His weight started increasing rapidly and his lust for his wife almost disappeared!
What was happening?
Well, it turns out that all that testosterone he got at the clinic – it was turning into feminizing estrogen.
In some cases, estrogen can jump from normal levels of 22 pnmol/L to a staggering 50 pnmol/L – which is in the extreme end of the normal range:
But luckily, my friend knew about DHT and how it’s so much better than testosterone.
This study on DHT found “an improvement in potency, libido, and frequency of intercourse not found in the placebo group…”
So if you want to safely increase your potency, libido, and sexual performance, you need DHT.
But you might be wondering where to get DHT…
You could try DHT supplements that can cause dry skin, changes in your bowel habits, dry mouth, muscle pain, increased thirst, increased urination, high blood pressure, stomach upset, loss of appetite, weight loss, or unusual taste in the mouth…
Or you can raise your DHT naturally and avoid all those nasty side-effects.
Your body can start making more DHT naturally with the help of a few simple activities (you don’t need to take anything).
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